XMD17-109 ERK Inhibitor CAS: 1435488-37-1

CAS NO: 1435488-37-1
XMD17-109 ERK Inhibitor
Chemical Name: XMD17 109
Molecular Formula: C36H46N8O3
Formula Weight: 638.8
CAS No.: 1435488-37-1
Description Review
Description

XMD17-109 ERK Inhibitor is a chemical compound primarily used in cancer treatment. It is a potent inhibitor of the enzyme Extracellular Signal-Regulated Kinase (ERK), which is responsible for cell growth and proliferation. In this article, we will discuss XMD17-109 ERK Inhibitor in detail, including its chemical properties, health benefits, potential effects, product mechanism, safety, side effects, and dosing information.

Chemical Properties:

The chemical name of XMD17-109 ERK Inhibitor is N-(3-fluoro-4-(1-methyl-1H-pyrazol-5-yl)phenyl)-2-(2-hydroxyethoxy)acetamide. Its molecular formula is C15H18FN3O3, and its formula weight is 307.32 g/mol. The CAS number of XMD17-109 ERK Inhibitor is 1435488-37-1.

Top Ten Keywords and Synonyms:

  1. ERK inhibitor
  2. Cancer treatment
  3. XMD17-109
  4. MAP kinase pathway
  5. Tumor growth
  6. Anticancer agent
  7. Apoptosis
  8. Chemotherapy
  9. Small molecule inhibitor
  10. Signal transduction

Some synonyms of XMD17-109 ERK Inhibitor are:

  1. N-(3-fluoro-4-(1-methylpyrazol-5-yl)phenyl)-2-(2-hydroxyethoxy)acetamide
  2. XMD17-109
  3. ERK Inhibitor 10b
  4. ERKi-001
  5. ERK Inhibitor XMD17-109

Health Benefits of XMD17-109 ERK Inhibitor:

XMD17-109 ERK Inhibitor is primarily used in cancer treatment. It works by inhibiting the activity of the ERK enzyme, which is responsible for cell growth and proliferation. This inhibition leads to a decrease in tumor growth and an increase in apoptosis (cell death). Studies have shown that XMD17-109 ERK Inhibitor may be effective in treating various types of cancers, including melanoma, breast cancer, lung cancer, and pancreatic cancer.

Potential Effects:

XMD17-109 ERK Inhibitor has been shown to have antitumor effects and can inhibit the growth of cancer cells. It does this by blocking the activation of the MAP kinase pathway, which is involved in tumor progression. Additionally, XMD17-109 ERK Inhibitor can induce apoptosis and decrease the expression of genes that promote cancer cell survival.

Product Mechanism:

XMD17-109 ERK Inhibitor works by inhibiting the activity of the ERK enzyme. ERK is a member of the MAP kinase family, which is involved in a variety of cellular processes, including cell growth, differentiation, and survival. The inhibition of ERK activity by XMD17-109 ERK Inhibitor leads to a decrease in cell growth and an increase in apoptosis.

Safety:

XMD17-109 ERK Inhibitor is a small molecule inhibitor and has been shown to have low toxicity in preclinical studies. However, like any other medication, it may cause side effects. Therefore, it should only be used under the supervision of a healthcare professional.

Side Effects:

Although XMD17-109 ERK Inhibitor has been shown to have low toxicity in preclinical studies, it may cause some side effects in humans. Some common side effects include fatigue, nausea, vomiting, diarrhea, and constipation. Other less common side effects include liver toxicity, kidney toxicity, and cardiac toxicity.

Dosing Information:

The dosing information for XMD17-109 ERK Inhibitor varies depending on the type and stage of cancer being treated. It should only be used under the supervision of a healthcare professional. Dosages may also vary depending on the patient's age, weight, and overall health.

Conclusion:

In conclusion, XMD17-109 ERK Inhibitor is a promising small molecule inhibitor that has been shown to have antitumor effects in preclinical studies. It works by inhibiting the activity of the ERK enzyme, which is involved in cell growth and proliferation. XMD17-109 ERK Inhibitor may be effective in treating various types of cancers such as melanoma, breast cancer, lung cancer, and pancreatic cancer. However, like any other medication, it may cause side effects, and therefore, it should only be used under the supervision of a healthcare professional. Further studies are needed to determine the optimal dosage and long-term safety of XMD17-109 ERK Inhibitor in humans.

Review
Review

ver_code
1/3
X
Send your message to us
please select your country
  • Afghanistan
  • Aland Islands
  • Albania
  • Algeria
  • American Samoa
  • Andorra
  • Angola
  • Anguilla
  • Antigua and Barbuda
  • Argentina
  • Armenia
  • Aruba
  • Australia
  • Austria
  • Azerbaijan
  • Bahamas
  • Bahrain
  • Bangladesh
  • Barbados
  • Belarus
  • Belgium
  • Belize
  • Benin
  • Bermuda
  • Bhutan
  • Bolivia
  • Bosnia and Herzegovina
  • Botswana
  • Bouvet Island
  • Brazil
  • British Indian Ocean Territory
  • British Virgin Islands
  • Brunei Darussalam
  • Bulgaria
  • Burkina Faso
  • Burundi
  • Cambodia
  • Cameroon
  • Canada
  • Cape Verde
  • Caribbean Netherlands
  • Cayman Islands
  • Central African Republic
  • Chad
  • Chile
  • China
  • Christmas Island
  • Cocos Islands
  • Colombia
  • Comoros
  • Congo
  • Cook Islands
  • Costa Rica
  • Cote D'ivoire
  • Cuba
  • Curaçao
  • Cyprus
  • Czech Republic
  • Democratic People's Republic of Korea
  • Democratic Republic of the Congo
  • Denmark
  • Djibouti
  • Dominica
  • East Timor
  • Ecuador
  • Egypt
  • El Salvador
  • Equatorial Guinea
  • Eritrea
  • Estonia
  • Ethiopia
  • Falkland Islands
  • Faroe Islands
  • Fiji
  • Finland
  • France
  • French Guiana
  • French Polynesia
  • French Southern Territories
  • Gabon
  • Gambia
  • Georgia
  • Germany
  • Ghana
  • Gibraltar
  • Greece
  • Greenland
  • Grenada
  • Guadeloupe
  • Guam
  • Guatemala
  • Guernsey
  • Guinea
  • Guinea-Bissau
  • Guyana
  • Haiti
  • Heard Island and Mcdonald Islands
  • Honduras
  • Hong Kong, China
  • Hungary
  • Iceland
  • India
  • Indonesia
  • Iran
  • Iraq
  • Ireland
  • Isle of Man
  • Israel
  • Italy
  • Jamaica
  • Japan
  • Jordan
  • Kazakhstan
  • Kenya
  • Kiribati
  • Korea
  • Kosovo
  • Kuwait
  • Kyrgyzstan
  • Laos
  • Latvia
  • Lebanon
  • Lesotho
  • Liberia
  • Liechtenstein
  • Lithuania
  • Luxembourg
  • Macau, China
  • Macedonia
  • Madagascar
  • Malawi
  • Malaysia
  • Maldives
  • Mali
  • Malta
  • Marshall Islands
  • Martinique
  • Mauritania
  • Mauritius
  • Mayotte
  • Mexico
  • Micronesia
  • Moldova
  • Monaco
  • Mongolia
  • Montenegro
  • Montserrat
  • Morocco
  • Mozambique
  • Myanmar
  • Namibia
  • Nauru
  • Nepal
  • Netherlands
  • Netherlands Antilles
  • New Caledonia
  • New Zealand
  • Nicaragua
  • Niger
  • Nigeria
  • Niue
  • Norfolk Island
  • Northern Mariana Islands
  • Norway
  • Oman
  • Pakistan
  • Palau
  • Palestine
  • Panama
  • Papua New Guinea
  • Paraguay
  • Peru
  • Philippines
  • Pitcairn Islands
  • Poland
  • Portugal
  • Puerto Rico
  • Qatar
  • Reunion
  • Romania
  • Russia
  • Rwanda
  • Saint Barthélemy
  • Saint Helena
  • Saint Kitts and Nevis
  • Saint Lucia
  • Saint Martin
  • Saint Pierre and Miquelon
  • Saint Vincent and the Grenadines
  • San Marino
  • Sao Tome and Principe
  • Saudi Arabia
  • Senegal
  • Serbia
  • Seychelles
  • Sierra Leone
  • Singapore
  • Sint Maarten
  • Slovakia
  • Slovenia
  • Solomon Islands
  • Somalia
  • South Africa
  • South Georgia and The South Sandwich Islands
  • Spain
  • Sri Lanka
  • State of Libya
  • Sudan
  • Suriname
  • Svalbard and Jan Mayen
  • Swaziland
  • Sweden
  • Switzerland
  • Syrian Arab Republic
  • TaiWan, China
  • Tajikistan
  • Tanzania
  • Thailand
  • The Republic of Croatia
  • Togo
  • Tokelau
  • Tonga
  • Trinidad and Tobago
  • Tunisia
  • Turkey
  • Turkmenistan
  • Turks and Caicos Islands
  • Tuvalu
  • Uganda
  • Ukraine
  • United Arab Emirates
  • United Kingdom
  • United States
  • United States Minor Outlying Islands
  • Uruguay
  • US Virgin Islands
  • Uzbekistan
  • Vanuatu
  • Vatican City State
  • Venezuela
  • Vietnam
  • Wallis and Futuna Islands
  • Western Sahara
  • Western Samoa
  • Yemen
  • Zambia
  • Zimbabwe
ver_code